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For research purposes only. Not for human consumption. Must be 18+. Verify COA

The anti-anxiety peptide that doesn't sedate you, doesn't numb you, and doesn't hook you.

Selank was developed alongside Semax by the Russian Academy of Sciences. It enhances GABA-A sensitivity and serotonin turnover in your limbic system — the same pathways targeted by benzodiazepines — but without the sedation, the memory fog, the tolerance buildup, or the withdrawal. Four-week clinical studies found zero dependency markers. Unlike Xanax, it also increases BDNF. Unlike SSRIs, it doesn't take 4-6 weeks to kick in. Twenty years of clinical use in Russia. Intranasal spray. Calm with clarity. Not drugged compliance.

COGNITIVE

Selank

Selank (Thr-Lys-Pro-Arg-Pro-Gly-Pro)

Selank is a synthetic analogue of the endogenous immunomodulatory peptide tuftsin, developed by the Russian Academy of Sciences. It is approved in Russia for anxiety and asthenic disorders. Unlike benzodiazepines, it provides anxiolytic effects via GABA-A and serotonin modulation without sedation, dependency, or cognitive blunting.

In Stock
Technical details
CAS number
129954-34-3
Molecular formula
C33H57N11O9
You might be wondering
You check it yourself. Every batch is tested by an independent, accredited third-party laboratory — the gold standard for peptide purity testing. Enter your batch number on our lab reports page and you'll see the exact purity percentage, mass confirmation, and endotoxin levels. No PDF → no product.

MECHANISM OF ACTION

Selank enhances GABA-A receptor sensitivity and modulates serotonin (5-HT) turnover in the limbic system — the primary mechanism behind its anxiolytic effect. It also increases BDNF in the hippocampus and prefrontal cortex, normalises IL-6 and other inflammatory cytokines, and stabilises enkephalin degradation, prolonging natural opioid signalling without addiction risk. The net effect is calm alertness, not sedation.

RESEARCH HIGHLIGHTS

1

Anxiolytic effect equivalent to benzodiazepines in rodent models with no sedation, ataxia, or memory impairment

Seredenin et al., 2008PubMed 18831451
2

Normalises IL-6 and other pro-inflammatory cytokines — distinct immunomodulatory mechanism from other anxiolytics

Kozlovskaya et al., 2002PubMed 12355388
3

Improves working memory and attention in anxious subjects — cognition-enhancing effect on top of anxiolytic base

Narkevich et al., 2008PubMed 18390064
4

No withdrawal syndrome observed after 4-week course; no physical or psychological dependency markers

Uchakina et al., 2008PubMed 18368936

RESEARCH PROTOCOLS

For laboratory use only. Not medical advice.

beginner
200mcg
once daily (intranasal)
Route: Oral / sublingual
Cycle: 2 weeks
Break: 1 week

Intranasal delivery. Effects felt within 15–30 minutes. Start in the morning or before a high-stress event. Many researchers report no adjustment period required.

intermediate
250mcg
twice daily (morning and evening)
Route: Oral / sublingual
Cycle: 4 weeks
Break: 2 weeks

Russian clinical standard. Pairs with Semax for balanced stress-and-focus protocol — Selank anchors the anxiolytic base, Semax amplifies executive function.

advanced
300mcg
three times daily
Route: Oral / sublingual
Cycle: 6 weeks
Break: 4 weeks

Used in clinical studies for generalised anxiety disorder. Monitor mood daily — reduce dose if emotional blunting observed.

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SAFETY & CONTRAINDICATIONS

Contraindications
  • Concurrent benzodiazepine use (potential additive GABA-A effect)
  • Pregnancy or breastfeeding
  • Severe hepatic or renal impairment
  • Known hypersensitivity to tuftsin or related peptides
Observed Effects

Excellent tolerability profile. Reported effects: mild nasal irritation (intranasal), transient drowsiness in first 2–3 days (resolves), appetite increase in some subjects. No cognitive impairment, no dependency, no withdrawal.

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For research and laboratory use only. Not intended for human consumption. Not for diagnostic or therapeutic purposes.